通讯作者: 朱何思, 605568434@qq.com

DOI：10.12201/bmr.202602.00069

Therapeutic Theoretical System of Emotional Diseases

• 摘要：[摘要] 情志病作为中医学独具特色的疾病分类范畴，以情志刺激诱发脏腑功能紊乱为核心病理环节。本研究以《黄帝内经》等中医经典文献为理论根基，整合现代神经生物学、心理学等多学科研究成果，系统构建涵盖20种情志病的完整治疗理论体系，明确五行与情志的对应关系及病态机制（乘、侮、亢、盗）、调节机制（胜、化、复、制、治、养），提出“调情志、和脏腑、顺五运”三维综合干预模式，按木、火、土、金、水五大脏腑系统阐释20种情志病的病机与干预方案，并借助现代研究验证传统理论科学性。研究证实，情志病的病机本质是神经-内分泌-免疫网络失衡，该体系为现代心身疾病防治提供理论指引与实践支撑，推动中医情志理论现代化转型。

  关键词： [关键词] 情志病; 五行学说; 病态机制; 调节方案; 现代验证

   

  Abstract: Abstract: Emotional diseases are a unique category of diseases in traditional Chinese medicine (TCM), with disorders of zang-fu functions induced by emotional stimulation as the core pathological link. Based on the theoretical foundation of TCM classic literature such as Huangdi Neijing, this study integrated the research results of modern neurobiology, psychology and other multidisciplinary fields, systematically constructed a complete therapeutic theoretical system covering 20 emotional diseases, clarified the corresponding relationship between the five elements and emotions, as well as the pathological mechanisms (Cheng, Wu, Kang, Dao) and regulatory mechanisms (Sheng, Hua, Fu, Zhi, Zhi, Yang). The three-dimensional comprehensive intervention model of "regulating emotions, harmonizing zang-fu organs, and following five movements" was proposed, and the pathogenesis and intervention schemes of 20 emotional diseases were explained according to the five zang-fu systems of wood, fire, earth, metal and water. The scientificity of traditional theories was verified with modern research. The study confirmed that the essential pathogenesis of emotional diseases is the imbalance of neuroendocrine-immune network, and this system provides theoretical guidance and practical support for the prevention and treatment of modern psychosomatic diseases, promoting the modern transformation of TCM emotional theory.

  Key words: Keywords: Emotional diseases; Five Elements Theory; Pathological mechanism; Regulatory scheme; Modern verification

  提交时间：2026-04-14

  版权声明：作者本人独立拥有该论文的版权，预印本系统仅拥有论文的永久保存权利。任何人未经允许不得重复使用。
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• 序号	提交日期	编号	操作
  1	2026-02-14	10.12201/bmr.202602.00069V1	下载

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